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Poster Abstracts - Z118
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Articles
Insights into the ER-mitochondria contact sites using protein interaction assay BioID
Identification
Aaltonen, Mari
INSIGHTS INTO THE ER-MITOCHONDRIA CONTACT SITES USING PROTEIN INTERACTION ASSAY BIOID
Pathophysiology of mitochondrial spinocerebellar ataxias in iPSC-derived neuronal cells
Identification
Äänismaa, Riikka
Pathophysiology of mitochondrial spinocerebellar ataxias in iPSC-derived neuronal cells
Bax-mediated mitochondrial membrane restructuring
Identification
Ader, Nicholas
BAX-MEDIATED MITOCHONDRIAL MEMBRANE RESTRUCTURING
Mitochondrial dysfunction is the key detrimental factor in the development of acute insulin resistance
Identification
Ahn, Ji Yun
MITOCHONDRIAL DYSFUNCTION IS THE KEY DETRIMENTAL FACTOR IN THE DEVELOPMENT OF ACUTE INSULIN RESISTANCE
Loss of mitochondrial protease YME1L in the muscle leads to fiber type switch and metabolic dysfunction
Identification
Ahola, Sofia
LOSS OF MITOCHONDRIAL PROTEASE YME1L IN THE MUSCLE LEADS TO FIBER TYPE SWITCH AND METABOLIC DYSFUNCTION
MicroRNA-210-3p improves the survival potential of adult mouse cardiac progenitors
Identification
Alonaizan, Rita
MICRORNA-210-3P IMPROVES THE SURVIVAL POTENTIAL OF ADULT MOUSE CARDIAC PROGENITORS
Age-dependent asymmetric apportioning of mitochondria during division determines cell fate
Identification
Amaral, Ana
AGE-DEPENDENT ASYMMETRIC APPORTIONING OF MITOCHONDRIA DURING DIVISION DETERMINES CELL FATE
CHCHD2 deficiency leads to mitochondrial dysfunction in human neuroblastoma SY-SY5Y cells
Identification
Amo, Taku
CHCHD2 DEFICIENCY LEADS TO MITOCHONDRIAL DYSFUNCTION IN HUMAN NEUROBLASTOMA SY-SY5Y CELLS
Studying Barth syndrome’s pathomechanism using high throughput screens in yeast
Identification
Antunes, Diana
STUDYING BARTH SYNDROME'S PATHOMECHANISM USING HIGH THROUGHPUT SCREENS IN YEAST
Mitochondrial dysfunction in cholesterol accumulated infected macrophages: Mycobacterium tuberculosis factors mediate persistence
Identification
Asalla, Suman
MITOCHONDRIAL DYSFUNCTION IN CHOLESTEROL ACCUMULATED INFECTED MACROPHAGES: MYCOBACTERIUM TUBERCULOSIS FACTORS MEDIATE PERSISTENCE
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