Neuroinflammation: Causes, Concepts and Consequences

Neuroinflammation comprises the interaction of two formidably complex organ systems. Two considerations underpin Keystone Symposia’s meetings on neuroinflammation: first that the communities of neuroscientists and immunologists must urgently formulate and deploy a joint set of concepts to promote mechanistic progress; and second, that neuroinflammation broadly conceived operates throughout life from embryogenesis through old age, with its consequences bearing on the most serious health concerns from autism and schizophrenia through dementia.

The roundtable will discuss:

Defining Neuroinflammation

Do we have a definition of neuroinflammation that encapsulates the different elements of immune-to-brain communication encompassing homeostasis to pathology in chronic neurodegenerative disease?

Disease Implications

What is the role of neuroinflammation throughout the aging process and in a wide range of neurodegenerative diseases including multiple sclerosis, Alzheimer’s Disease and Parkinson’s?

New Ways to Study the Brain

How can we better study neuroinflammation in the cells of the brain and are there ways to collaborate in developing new imaging and sampling technologies and datasets, in vivo and postmortem, that will advance this field?

Pre-Competitive Research

What are ways that academic and industrial scientists from different backgrounds can collaborate on pre-competitive research?

The panel will touch on some of the most important learnings and highlights from the Keystone Symposia four-day meeting in June 2017 on Neuroinflammation. It will also brainstorm ideas for subsequent meetings, including the June 2018 symposium on “New Frontiers in Neuroinflammation: What Happens When CNS and Periphery Meet” which is being organized by Drs. Marco Prinz, Jonathan Kipnis and Irene Knuesel.

Event Hashtag: #VKSneuroinf17

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